By Peter C. Nowell (auth.), Frederick F. Becker (eds.)
This sequence of books makes an attempt to give, in a entire demeanour, the sector of oncology divided into 3 significant components; etiology, biology, and remedy. those books should still function landmarks within the swiftly increasing experimental and scientific "universe" of this box. to a few, they are going to be introductory; to others, a precis; for all, serious reviews at the futuI'e of analysis. In l'ecognition of the problems inherent in trying to pause and replicate whereas experimental information emerge with ever-increasing rapidity, the shows take the shape of overviews instead of studies. the place attainable, an historic viewpoint on observations and experimentation which resulted in our current knowing is gifted, the cutting-edge in strategy and process is reviewed, and the gaps in wisdom and in process are indicated. the purpose all through is integration-using the findings from one strategy for comparability with others. The great growth of curiosity in oncology as a medical-biological self-discipline influenced the booklet of those volumes. This growth, good warranted by way of the influence of oncology on human morbidity, has been characterised through at the very least 3 phenomena. First, there was an immense bring up in funds and manpower dedicated to the research and remedy of malignancy. That the study has turn into increasingly more "directed" or program-oriented signs the curiosity of these past the clinical group within the administration of the eHort. moment, expanding numbers of scholars are getting into the sector of oncology as their significant education program.
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Additional info for Cancer. A Comprehensive Treatise: Volume 1. Etiology: Chemical and Physical Carcinogenesis
8:61. , MILLER, O. ) 223:363. HAUSCHKA, T. , 1961, The chromosome in ontogeny and oncogeny, Cancer Res. 21:957. , MCCAW, B. , AND KOLER, R. , 1973, Ataxia-telangiectasia-Clonal growth of translocation lymphocytes, New Engl. J. Med. 289:286. ) 231 :511. Hsu, T. , 1961, Chromosomal evolution in cell populations, Int. Rev. Cytol. 12:69. HUANG, C. , SMITH, R. , AND OSUNKOYA, B. , 1970, Revaluation of relationship between C chromosome marker and Epstein-Barr virus, J. Natl. Cancer Inst. 45:815. , TOUGH, I.
J. Haematol. 23:283. FITZGERALD, P. , 1965, Abnormal length of the small acrocentric chromosomes in chronic lymphocytic leukemia, Cancer Res. 25:1094 FITZGERALD, P. , CROSSEN, P. , ADAMS, A. , SHARMAN,C. , AND GUNZ, F. , 1966, Chromosome studies in familial leukemia, J. Med. Genet. 3:96. FITZGERALD, P. , CROSSEN, P. , AND HAMER, J. , 1973, Abnormal karyotypic clones in human acute leukemia: Their nature and significance, Cancer 31:1069. FORD, C. , 1966, The use of chromosome markers, in: Tissue Grafting and Radiation (H.
Chromosome changes of particular interest have been reported in three of the less common lymphoproliferative disorders: multiple myeloma, Waldenstrom's macroglobulinemia, and Burkitt's lymphoma. In a significant proportion of multiple myeloma patients, abnormal chromosomes have been observed in some of the dividing cells of the bone marrow, presumably derived from a chromosome in group D (chromosomes 13-15) in some cases and resembling a large group-A member in others. Similarly, a number of cases of Waldenstrom's macroglobulinemia have been described in which the karyotype was characterized by a large marker chromosome, again apparently derived from one of the group-A chromosomes (Nos.
Cancer. A Comprehensive Treatise: Volume 1. Etiology: Chemical and Physical Carcinogenesis by Peter C. Nowell (auth.), Frederick F. Becker (eds.)