By H. Jellinek (auth.), H. Jellinek (eds.)

ISBN-10: 1461587115

ISBN-13: 9781461587118

ISBN-10: 1461587131

ISBN-13: 9781461587132

This monograph summarizes the result of a fifteen-year vascular pathology study venture backed by way of the Hungarian Academy of Sciences. the staff who've participated during this undertaking are indebted to the Academy for financing huge investigations and visits to institutes in another country, the goals of which have been to resolve sure difficulties through foreign cooperation. the consequences awarded by way of the collective attempt of study groups, each one learning right here were completed a specific challenge, yet operating jointly to an analogous finish. Mutual dialogue of findings and a typical alternate of data among the learn teams have contributed to the development of the investigations. typical e-book of information and mutual feedback ensured that the findings have been uniformly interpreted. it truly is was hoping that the elemental objective of our long term undertaking, i. e. the choice of the ideal method of the complicated challenge of vascular pathology, has been completed. the recent procedure effects from the popularity that for the reason that arteriosclerosis ultimately ends up in different types of vascular damage, realization could be focussed at the occasions previous the improvement of the lesions instead of at the lesions themselves. This declare has been proven by means of numerous version experiments during which vascular harm inflicted below varied stipulations uniformly ended in morphologi­ cally validated arteriosclerotic swap. the new elevate within the prevalence of civi­ lization ailments of the vascular approach has ended in a world-wide growth of arteriosclerosis research.

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Example text

The microfibrils were closely associated with the elastic clumps, lying at their margins (Plate XV/65-67). Elastic fibres developing at the external part of the media began to lose PTA-binding capacity (Plate XVj68) after 9 days, and completely mature fibres with exclusively marginal PTA-affinity were already seen after 14 days (Plate XVIIIj78). Both light- and electron-microscopic observations suggest that elastic fibre genesis proceeds towards the vascular lumen and reaches the extracellular area below the endothelial cell row by the 17th day.

Intimal proliferation develops only secondarily, partly through phagocytosis of the fibrinoid, partly through mobilization and migration of smooth muscle cells. Apart from the fundamental identity of the primary response, the modifying effect of the structural differences also comes into display. 1. The most conspicuous modifications have been observed in connection with the elastic elements. In small arteries, in which the IEL represents a single continuous elastic structure, necrotic medial smooth muscle cells and inflowing plasma together formed a media fibrinoid (see Chapter 9).

The development of granulation was probably related to the extravasation of plasma into the perivascular space.

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Arterial Lesions and Arteriosclerosis by H. Jellinek (auth.), H. Jellinek (eds.)


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