By H. Kalant (auth.), Milton M. Gross (eds.)
The contributions to this quantity essentially point out the momentum, caliber, liveliness and variety of the study attempt being di rected towards deepening our figuring out of tqe alcohol withdrawal syndrome. This zone of analysis has received expanding curiosity and a spotlight to the purpose the place it appeared average to dedicate a spe cial part to it on the thirtieth overseas Congress for Alcoholism and Drug Dependence in Amsterdam in September, 1972. Our aim used to be to proportion our latest findings and stimulate others to affix within the attempt. With few exceptions, the papers during this e-book current new info. It were was hoping that the quantity would seem through the top of 1972. although, the fervour of a number of the individuals to increase their investigations made this goal date most unlikely. a number of of the papers weren't awarded within the part yet have been offered in different places within the Congress. although, as a result of their specific relevance to the subject they've been integrated with the beneficiant consent of the authors.
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Additional resources for Alcohol Intoxication and Withdrawal I: Experimental Studies
1971. The alteration of brain metabolism by narcotic analgesic drugs, in Handbook of Neurochemistry, Vol. VI (A. ) pp. 479 - 508, Plenum Press, New York London. Cohen, G. , 1970. Alkaloids from catecholamines in adrenal tissue: Possible role in alcoholism, Science 167: 1749 - 1751. , 1970. Augmentation of alkaloid formation from dopamine by alcohol and acetaldehyde in vitro, J. Pharmacol. Exp. Ther. 174: 401 - 412. W. , 1970. Experimentally induced ethanol dependence in rhesus monkeys, J. Pharmacol.
Similarly, alterations in the peripheral metabolism of norepinephrine, as well as serotonin have ensued after the systemic administration of acetaldehyde in rats (11) or ethanol in humans (12,13). , those derived from serotonin and dopamine) favor their metabolism by the oxidative pathway in brain tissue. Therefore, the inhibition of the oxidative pathway would most probably l~ad to an increase in the steady state level of these particular aldehydes. The metabol ism of such aldehydes by the reductase, although increased, would most probably not be adequate to prevent an increase in the intracellular concentration of these aldehydes and their participation in other reactions.
Lajtha, A. , 1971. Protein turnover, in Handbook of Neurochemistry, Vol. VB (A. ) pp. 551 - 629. Plenum Press, New York - London. J. , 1969. Acquisition and loss of tolerance to ethanol by the rat, J. Pharmacol. Exp. Ther. 168: 244 - 250. Lindbohm, R. , 1962. Changes in respiration of rat brain cortex slices induced by some aliphatic alcohols, Acta Pharmacol. Toxicol. 19: 53 - 58. , 1971. Some neurochemical aspects of convulsions, in Handbook of Neurochemistry, Vol. VI (A. ) pp. 63 102, Plenum Press, New York - London.
Alcohol Intoxication and Withdrawal I: Experimental Studies by H. Kalant (auth.), Milton M. Gross (eds.)